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Visual extinction
Visual extinction is a neurological disorder which occurs following damage to the parietal lobe of the brain. It is similar to, but distinct from, hemispatial neglect. Visual extinction has the characteristic symptom of difficulty to perceive contralesional stimuli when presented simultaneously with an ipsilesional stimulus, but the ability to correctly identify them when not presented simultaneously. Under simultaneous presentation, the contralesional stimulus is apparently ignored by the patient, or extinguished. This deficiency may lead to difficulty on behalf of the patient with processing the stimuli’s 3D position.〔Vuilleumier, PO, Rafal, RD (2000). "A systematic study of visual extinction: between- and within-field deficits of attention in hemispatial neglect". ''Brain'' 123,1263-1279.()〕〔"Neuroexam.com - Vision (CN II)." neuroexam.com - An interactive online guide to the neurologic examination. 30 Sep. 2009 ()〕
==History==
Visual Extinction is the result of unilateral cerebral damage〔Pavlovskaya, Marina, Sagi, Dov, Soroker, Nachum, Ring Haim (1997). "Visual extinction and cortical connectivity in human vision". ''Cognitive Brain Research'' 6(2), 159-162.()〕 and has always been poorly understood. Researchers have been studying visual extinction in great depth since the 1990s. It has since been commonly associated with damage to the right hemisphere of the brain. Studies have suggested that visual extinction may be a result of sensory imbalance. This imbalance is due to weak or delayed afferent inputs in the hemisphere affected by the extinction.〔
Research done by Pavlovskaya, Sagi, Soroker and Ring show that visual extinction is dependent on simple stimuli properties. These properties are thought to reflect connectivity constraints during the early steps of visual processing.〔
抄文引用元・出典: フリー百科事典『 ウィキペディア（Wikipedia）』
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